Contagion explores cultural responses of infectious diseases and their biomedical management over the nineteenth and twentieth centuries. It also investigates the use of 'contagion' as a concept in postmodern research.
Looks at the connection between trade and disease, tracing the plagues that swept through Eurasia in the fourteenth century and exposes the weaknesses in the current public health system that make our world susceptible to a pandemic.
An argument that contagion is the most significant risk facing the financial system and that Dodd¬Frank has reduced the government's ability to respond effectively. The Dodd–Frank Act of 2010 was intended to reform financial policies in order to prevent another massive crisis such as the financial meltdown of 2008. Dodd–Frank is largely premised on the diagnosis that connectedness was the major problem in that crisis—that is, that financial institutions were overexposed to one another, resulting in a possible chain reaction of failures. In this book, Hal Scott argues that it is not connectedness but contagion that is the most significant element of systemic risk facing the financial system. Contagion is an indiscriminate run by short-term creditors of financial institutions that can render otherwise solvent institutions insolvent. It poses a serious risk because, as Scott explains, our financial system still depends on approximately $7.4 to $8.2 trillion of runnable and uninsured short-term liabilities, 60 percent of which are held by nonbanks. Scott argues that efforts by the Federal Reserve, the FDIC, and the Treasury to stop the contagion that exploded after the bankruptcy of Lehman Brothers lessened the economic damage. And yet Congress, spurred by the public's aversion to bailouts, has dramatically weakened the power of the government to respond to contagion, including limitations on the Fed's powers as a lender of last resort. Offering uniquely detailed forensic analyses of the Lehman Brothers and AIG failures, and suggesting alternative regulatory approaches, Scott makes the case that we need to restore and strengthen our weapons for fighting contagion.
As the outbreak of a new and deadly form of coronavirus dominates headlines and triggers fear and global recession, now is a good time to reflect on the history and science of transmissible diseases. Behind every disease is a story, from the natural history of the disease and its course in the individual, to the tale of the disease's description, discovery and treatment. From the impact of tuberculosis on English dynastic history to the makeup of our DNA; from the deadliest plagues of the ancient world to twenty-first century pandemics; and from the ravages of the Black Death to the discovery of antibodies, transmissible diseases have an incredible variety of tales to tell. Contagion explores some of the most notorious, grisly, and pernicious communicable diseases in history, revealing their hidden stories. In addition to discussing their symptoms, causes, prevention, and treatment, Richard Gunderman also discusses their impact on notable figures in history,nfrom soldiers to monarchs; the extraordinary contributions of the scientists and physicians who battled them; as well as their impacts on world history and human evolution. Here are the exploits of Edward Jenner, who invented the first vaccine; John Snow, the first person to study disease scientifically; Louis Pasteur, who established the germ theory of infection, along with a myriad other remarkable stories in the never ending struggle between humanity and disease. The narrative is brought right up to date with the desperate battle to stem the Covid-19 pandemic and discover a vaccine. Renowned medical expert Dr Richard Gunderman shows how disease has shaped the evolution of our species and, if we don't take the proper steps, may yet threaten our very existence on this planet.
Expanding our understanding of contagion beyond the typical notions of infection and pandemics, this book widens the field to include the concept of biosocial epidemics. The chapters propose varied and detailed answers to questions about epidemics and their contagious potential for specific infections and non-infectious conditions. Together they explore how inseparable social and biological processes configure co-existing influences, which create epidemics, and in doing so stress the role of social inequality in these processes. The authors compellingly show that epidemics do not spread evenly in populations or through simple coincidental biological contagion: they are biosocially structured and selective, and happen under specific economic, political and environmental conditions. This volume illustrates that an understanding of biosocial factors is vital for ensuring effective strategies for the containment of epidemics.
This paper presents evidence that spillovers through bank lending, as opposed to trade linkages and country characteristics, can help explain contagion. We construct a measure of competition for bank funds and find evidence in favor of a common lender effect in the Mexican, Thai, and Russian crises, after controlling for macroeconomic fundamentals. The results are quite robust to the definition of the finance indicator. In the case of the Asian crisis, results are not always robust to the inclusion of trade competition, reflecting the high correlation between competition for funds and trade.